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电子期刊 >> 正文 |
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摘要：
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临床医生在实践工作中,很早就发现脑缺血伴发炎症反应,但直到近几年才对其有了较为清楚的认识.脑缺血时白细胞浸润发生在缺血后6～24小时内,促使白细胞向血管外迁移的细胞因子产生在脑缺血的初始部位.脑动脉阻断后即使立即再通,脑组织的缺血损伤也不能完全恢复正常,而且不能完全挽救由继发损伤造成的脑缺血组织的继续扩大.
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作者简介：
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参考文献：
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Hart RG.FosteRJW.LutheRMF Stroke in infective endocarditis 1990 ZiegleRCM.Schwarz W.Grau AJ Odontogenic focus as the etiology of cerebral ischemia 1998 Uza G Isurinary infection asarisk factoRfoRischemic cerebrovasculaRdisease 1997 Hallenbeck JM.Dutka AJ.kockanek PM Stroke riskfactors prepare rat brainstem fissues foRmodified local shwartzman reaction 1988 Grau AJ.Baggle F.BecheRH Recent bacterial and viral infection is a rist factoRfoRcerebrovas CulaRischemia\^clinical and biochemical studies 1998 Brwes MP.Rothlein R.Fegansc Monoclond antibodies preventing leukocyte activation reduce experimental neurologic injury and enhance efficacy of thrombolytic therapy 1995 HartlR.SchureRL.Schmid-Schonbein GW Fxpenimental antilenkocyte intenventions in cerebral ischemia\ 1996
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